Zn2+ inhibits the anion transport activity of band 3 by binding to its cytoplasmic tail.

نویسندگان

  • Y P Tu
  • H Xu
چکیده

Zn2+ can induce a conformational change of Band 3 with concomitant inhibition of its anion transport activity of human erythrocyte membrane vesicles only from the cytoplasmic side. The Zn2+ inhibition exhibits a dose-dependent manner with an apparent half maximal concentration of 50 microM ZnCl2 and can be reversed by 0.5 mM EDTA, but not by 1 mM dithiothreitol. The Zn2+ effect is specific and no similar inhibitory action could be observed by other divalent cations (Cu2+, Mn2+, Mg2+ or Sr2+).

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Persistence of external chloride and DIDS binding after chemical modification of Glu-681 in human band 3.

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ACELL October 46/4

Bahar, Sonya, Christopher T. Gunter, Cheryl Wu, Scott D. Kennedy, and Philip A. Knauf. Persistence of external chloride and DIDS binding after chemical modification of Glu-681 in human band 3. Am. J. Physiol. 277 (Cell Physiol. 46): C791–C799, 1999.—Although its primary function is monovalent anion exchange, the band 3 protein also cotransports divalent anions together with protons at low pH. T...

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عنوان ژورنال:
  • Bioscience reports

دوره 14 4  شماره 

صفحات  -

تاریخ انتشار 1994